authors = dictio['editor']

code/cit.py

@@ -215,7 +215,10 @@ def bookHTML(dictio, x, out):
     out.write("<li typeof=\"schema:Book\" role=\"doc-biblioentry\" property=\"schema:citation\" id=\"desc-" + str(x) + "\">"+ "\n")
    # out.write("\t" + "<span property=\"schema:author\" typeof=\"schema:Organisation\">"+ "\n")
     print("Just a sec, separating authors...")
-    authors = dictio['author']
+    if 'authors' in dictio:
+        authors = dictio['author']
+    elif 'editor' in dictio:
+        authors = dictio['editor']
     makeauthors(authors, out)
    # out.write("\t" + "\t" +"<span property=\"schema:Name\"> " + aut + "</span>."+ "\n")
    # out.write("\t" +"</span>"+ "\n")

code/methods.bib

+[1]
+@article{article,
+	title        = {Die Entwicklung der Patch-Clamp-Technik},
+	author       = {Roth, F. C., Numberger, M., and Draguhn, A.},
+	year         = 2023,
+	month        = {{}},
+	journal      = {Springer eBooks},
+	volume       = {{}},
+	pages        = {1--14},
+	doi          = {10.1007/978-3-662-66053-9}
+}
+[2]
+@book{dallas_patch_2021,
+	title        = {Patch clamp electrophysiology: methods and protocols},
+	shorttitle   = {Patch clamp electrophysiology},
+	year         = 2021,
+	publisher    = {Humana Press},
+	address      = {New York},
+	series       = {Methods in molecular biology},
+	number       = 2188,
+	isbn         = {978-1-07-160818-0 978-1-07-160820-3},
+	language     = {en},
+	editor       = {Dallas, Mark and Bell, Damian}
+}
+[3]
+@article{PRIEL20073893,
+	title        = {
+		Ionic Requirements for Membrane-Glass Adhesion and Giga Seal Formation in
+		Patch-Clamp Recording
+	},
+	author       = {
+		Avi Priel and Ziv Gil and Vincent T. Moy and Karl L. Magleby and Shai D.
+		Silberberg
+	},
+	year         = 2007,
+	journal      = {Biophysical Journal},
+	volume       = 92,
+	number       = 11,
+	pages        = {3893--3900},
+	doi          = {10.1529/biophysj.106.099119},
+	issn         = {0006-3495},
+	url          = {https://www.sciencedirect.com/science/article/pii/S000634950771189X},
+	abstract     = {
+		Patch-clamp recording has revolutionized the study of ion channels,
+		transporters, and the electrical activity of small cells. Vital to this
+		method is formation of a tight seal between glass recording pipette and cell
+		membrane. To better understand seal formation and improve practical
+		application of this technique, we examine the effects of divalent ions,
+		protons, ionic strength, and membrane proteins on adhesion of membrane to
+		glass and on seal resistance using both patch-clamp recording and atomic
+		force microscopy. We find that H+, Ca2+, and Mg2+ increase adhesion force
+		between glass and membrane (lipid and cellular), decrease the time required
+		to form a tight seal, and increase seal resistance. In the absence of H+
+		(10−10M) and divalent cations (<10−8M), adhesion forces are greatly reduced
+		and tight seals are not formed. H+ (10−7M) promotes seal formation in the
+		absence of divalent cations. A positive correlation between adhesion force
+		and seal formation indicates that high resistance seals are associated with
+		increased adhesion between membrane and glass. A similar ionic dependence of
+		the adhesion of lipid membranes and cell membranes to glass indicates that
+		lipid membranes without proteins are sufficient for the action of ions on
+		adhesion.
+	}
+}
+[4]
+@article{10.3389/fphar.2017.00195,
+	title        = {
+		Development of Automated Patch Clamp Technique to Investigate CFTR Chloride
+		Channel Function
+	},
+	author       = {
+		Billet, Arnaud  and Froux, Lionel  and Hanrahan, John W.  and Becq, Frederic
+	},
+	year         = 2017,
+	journal      = {Frontiers in Pharmacology},
+	volume       = 8,
+	doi          = {10.3389/fphar.2017.00195},
+	issn         = {1663-9812},
+	url          = {
+		https://www.frontiersin.org/journals/pharmacology/articles/10.3389/fphar.2017.00195
+	}
+}
+New5.
+@article{DuBridge_Tang_Hsia_Leong_Miller_Calos_1987,
+	title        = {
+		Analysis of mutation in human cells by using an Epstein-Barr virus shuttle
+		system.
+	},
+	author       = {
+		DuBridge, R B and Tang, P and Hsia, H C and Leong, P M and Miller, J H and
+		Calos, M P
+	},
+	year         = 1987,
+	month        = jan,
+	journal      = {Molecular and Cellular Biology},
+	volume       = 7,
+	number       = 1,
+	pages        = {379–387},
+	issn         = {0270-7306},
+	abstractnote = {
+		We developed highly sensitive shuttle vector systems for detection of
+		mutations formed in human cells using autonomously replicating derivatives of
+		Epstein-Barr virus (EBV). EBV vectors carrying the bacterial lacI gene as the
+		target for mutation were established in human cells and later returned to
+		Escherichia coli for rapid detection and analysis of lacI mutations. The
+		majority of the clonal cell lines created by establishment of the lacI-EBV
+		vector show spontaneous LacI- frequencies of less than 10(-5) and are
+		suitable for studies of induced mutation. The ability to isolate clonal lines
+		represents a major advantage of the EBV vectors over transiently replicating
+		shuttle vectors (such as those derived from simian virus 40) for the study of
+		mutation. The DNA sequence changes were determined for 61 lacI mutations
+		induced by exposure of one of the cell lines to N-nitroso-N-methylurea. A
+		total of 33 of 34 lacI nonsense mutations and 26 of 27 missense mutations
+		involve G X C to A X T transitions. These data provide support for the
+		mutational theory of cancer.
+	}
+}
+[New6].
+@article{Qin_Zhang_Clift_Hulur_Xiang_Ren_Lahn_2010,
+	title        = {
+		Systematic Comparison of Constitutive Promoters and the Doxycycline-Inducible
+		Promoter
+	},
+	author       = {
+		Qin, Jane Yuxia and Zhang, Li and Clift, Kayla L. and Hulur, Imge and Xiang,
+		Andy Peng and Ren, Bing-Zhong and Lahn, Bruce T.
+	},
+	year         = 2010,
+	month        = may,
+	journal      = {PLOS ONE},
+	publisher    = {Public Library of Science},
+	volume       = 5,
+	number       = 5,
+	pages        = {e10611},
+	doi          = {10.1371/journal.pone.0010611},
+	issn         = {1932-6203},
+	abstractnote = {
+		Constitutive promoters are used routinely to drive ectopic gene expression.
+		Here, we carried out a systematic comparison of eight commonly used
+		constitutive promoters (SV40, CMV, UBC, EF1A, PGK and CAGG for mammalian
+		systems, and COPIA and ACT5C for Drosophila systems). We also included in the
+		comparison the TRE promoter, which can be activated by the rtTA
+		transcriptional activator in a doxycycline-inducible manner. To make our
+		findings representative, we conducted the comparison in a variety of cell
+		types derived from several species. We found that these promoters vary
+		considerably from one another in their strength. Most promoters have fairly
+		consistent strengths across different cell types, but the CMV promoter can
+		vary considerably from cell type to cell type. At maximal induction, the TRE
+		promoter is comparable to a strong constitutive promoter. These results
+		should facilitate more rational choices of promoters in ectopic gene
+		expression studies.
+	},
+	language     = {en}
+}
+new7.
+@article{Bulcaen_Kortleven_Liu_Maule_Dreano_Kelly_Ensinck_Thierie_Smits_Ciciani_e,
+	title        = {
+		Prime editing functionally corrects cystic fibrosis-causing CFTR mutations in
+		human organoids and airway epithelial cells
+	},
+	author       = {
+		Bulcaen, Mattijs and Kortleven, Phéline and Liu, Ronald B. and Maule, Giulia
+		and Dreano, Elise and Kelly, Mairead and Ensinck, Marjolein M. and Thierie,
+		Sam and Smits, Maxime and Ciciani, Matteo and Hatton, Aurelie and Chevalier,
+		Benoit and Ramalho, Anabela S. and Casadevall i Solvas, Xavier and Debyser,
+		Zeger and Vermeulen, François and Gijsbers, Rik and Sermet-Gaudelus, Isabelle
+		and Cereseto, Anna and Carlon, Marianne S.
+	},
+	year         = 2024,
+	month        = may,
+	journal      = {Cell Reports Medicine},
+	pages        = 101544,
+	doi          = {10.1016/j.xcrm.2024.101544},
+	issn         = {2666-3791},
+	abstractnote = {
+		Prime editing is a recent, CRISPR-derived genome editing technology capable
+		of introducing precise nucleotide substitutions, insertions, and deletions.
+		Here, we present prime editing approaches to correct L227R- and N1303K-CFTR,
+		two mutations that cause cystic fibrosis and are not eligible for current
+		market-approved modulator therapies. We show that, upon DNA correction of the
+		CFTR gene, the complex glycosylation, localization, and, most importantly,
+		function of the CFTR protein are restored in HEK293T and 16HBE cell lines.
+		These findings were subsequently validated in patient-derived rectal
+		organoids and human nasal epithelial cells. Through analysis of predicted and
+		experimentally identified candidate off-target sites in primary stem cells,
+		we confirm previous reports on the high prime editor (PE) specificity and its
+		potential for a curative CF gene editing therapy. To facilitate future
+		screening of genetic strategies in a translational CF model, a machine
+		learning algorithm was developed for dynamic quantification of CFTR function
+		in organoids (DETECTOR: “detection of targeted editing of CFTR in
+		organoids”).
+	}
+}
+new8.
+@article{Ensinck_Deeersmaecker_Heylen_Ramalho_Gijsbers_Far,
+	title        = {
+		Phenotyping of Rare CFTR Mutations Reveals Distinct Trafficking and
+		Functional Defects
+	},
+	author       = {
+		Ensinck, Marjolein and De Keersmaecker, Liesbeth and Heylen, Lise and
+		Ramalho, Anabela S. and Gijsbers, Rik and Farré, Ricard and De Boeck, Kris
+		and Christ, Frauke and Debyser, Zeger and Carlon, Marianne S.
+	},
+	year         = 2020,
+	month        = mar,
+	journal      = {Cells},
+	volume       = 9,
+	number       = 3,
+	pages        = 754,
+	doi          = {10.3390/cells9030754},
+	issn         = {2073-4409},
+	abstractnote = {
+		Background. The most common CFTR mutation, F508del, presents with multiple
+		cellular defects. However, the possible multiple defects caused by many rarer
+		CFTR mutations are not well studied. We investigated four rare CFTR mutations
+		E60K, G85E, E92K and A455E against well-characterized mutations, F508del and
+		G551D, and their responses to corrector VX-809 and/or potentiator VX-770.
+		Methods. Using complementary assays in HEK293T stable cell lines, we
+		determined maturation by Western blotting, trafficking by flow cytometry
+		using extracellular 3HA-tagged CFTR, and function by halide-sensitive YFP
+		quenching. In the forskolin-induced swelling assay in intestinal organoids,
+		we validated the effect of tagged versus endogenous CFTR. Results. Treatment
+		with VX-809 significantly restored maturation, PM localization and function
+		of both E60K and E92K. Mechanistically, VX-809 not only raised the total
+		amount of CFTR, but significantly increased the traffic efficiency, which was
+		not the case for A455E. G85E was refractory to VX-809 and VX-770 treatment.
+		Conclusions. Since no single model or assay allows deciphering all defects at
+		once, we propose a combination of phenotypic assays to collect rapid and
+		early insights into the multiple defects of CFTR variants.
+	},
+	language     = {eng}
+}
+[new9]
+@misc{ignatova2023,
+	title        = {
+		Research Group Ignatova at the Institute of Biochemistry and Molecular
+		Biology
+	},
+	author       = {Zoya Ignatova},
+	year         = 2023,
+	url          = {https://www.chemie.uni-hamburg.de/institute/bc/arbeitsgruppen/ignatova.html},
+	note         = {Accessed: 20 August 2024},
+	institution  = {University of Hamburg}
+}
+new10.
+@book{Mennella_2024,
+	title        = {Cilia: methods and protocols},
+	year         = 2024,
+	publisher    = {Humana Press},
+	address      = {New York, NY},
+	isbn         = {978-1-07-163507-0},
+	abstractnote = {
+		This volume covers the latest advancements in the study of ciliary
+		complexity. Protocols cover genomic, proteomic, imaging, and functional
+		analysis of different ciliated tissues and their wide applicability in cilia
+		biology. Chapters in this book primarily focus on methods to study
+		multiciliated cells, and discuss topics such as SARS-CoV-2 infections of
+		human primary nasal multiciliated epithelial cells; expansion microscopy of
+		ciliary proteins; live-imaging centriole amplification in mouse brain
+		multiciliated cells; biophysical properties of cilia motility; and
+		mucociliary transport device construction. Written in the highly successful
+		Methods in Molecular Biology series format, chapters include introductions to
+		their respective topics, lists of the necessary materials and reagents,
+		step-by-step, readily reproducible laboratory protocols, and tips on
+		troubleshooting and avoiding known pitfalls. Cutting-edge and thorough,
+		Cilia: Methods and Protocols is a valuable resource for researchers who are
+		interested in learning more about this developing field.
+	},
+	language     = {eng}
+}

code/output.txt

 {/*<!-- Citation num 1--> */}
 <li typeof="schema:ScolarlyArticle" role="doc-biblioentry" property="schema:citation" id="desc-1">
+	<span property="schema:author" typeof="schema:Person">
+		<span property="schema:Name"> Roth, F. C.</span>
+		<span property="schema:Name"> Draguhn, A.</span>
+	</span>
+	<span property="schema:name">&nbsp;Die Entwicklung der Patch-Clamp-Technik</span>. 
+	<i property="schema:publisher" typeof="schema:Organization"> Springer eBooks</i>
+	<b property="issueNumber" typeof="PublicationIssue"> </b>
+	,&nbsp;<span property="schema:pageBegin"> 1</span>-<span property="schema:pageEnd">14</span>&nbsp;
+	(<time property="schema:datePublished" datatype="xsd:gYear" dateTime=" 2023">2023</time>).
+	<a className="doi" href="https://doi.org/10.1007/978-3-662-66053-9"> doi: 10.1007/978-3-662-66053-9</a>
+</li>
+
+{/*<!-- Citation num 2--> */}
+<li typeof="schema:Book" role="doc-biblioentry" property="schema:citation" id="desc-2">