Update wiki/pages/proof-of-concept.html

wiki/pages/proof-of-concept.html

@@ -343,7 +343,7 @@ project.{% endblock %}
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             <p class="content">To enable effective glucocorticoid sensing in mammalian cells, we engineered a synthetic
               transcriptional factor Tet<sub>stress</sub> which rewires glucocorticoid signal to a
-              P<sub>tetO7</sub>-driven ectopic gene expression (<strong>Figure 1a</strong>). Among the
+              P<sub>TetO7</sub>-driven ectopic gene expression (<strong>Figure 1a</strong>). Among the
               Tet<sub>stress</sub> variants we developed, the GR<sub>LBD</sub>-TetR configuration and the
               GR<sub>LBD</sub>-NES-TetR demonstrated optimal glucocorticoid responsiveness (see our <a
                 href="https://2022.igem.wiki/nudt-china/engineering">Engineering Success</a> page for the details of the