diff --git a/src/contents/results.tsx b/src/contents/results.tsx
index 09bb27741390d2c6aa179561e1a6fab8837a587c..c62074b8ce82e28d6b992d5634665e92db27fbba 100644
--- a/src/contents/results.tsx
+++ b/src/contents/results.tsx
@@ -108,12 +108,6 @@ export function Results() {
                 <p>According to a review in Journal of Hepatology[2] , the physiological level of blood ammonia is around 35~50μM, a level of 120μM is associated with high risk of death in HE patients.</p>
                 <p>As shown in Figure 2b, the difference between EcN_vector and EcN_GS with lower NH4Cl concentration such as 5μM decreased and was even not significant with 0.5μM. This indicates that the GS enzyme requires a rather high level NH4Cl to reach the most efficient status. Therefore, when the level of ammonia is in a normal range, the metabolic module is not likely to cause a significant decrease in ammonia, which might be useful in other metabolic cycles. </p>
                 <p>These results could allay some safety concerns of expressing GS enzymes to some extent, but more animal experiments are essential for further evaluation.</p>
-                <img 
-                src=""
-                alt="result"
-                className="responsive-img"
-              />
-               <figcaption className='caption'></figcaption>
               </Element>