diff --git a/src/data/drug-data.tsx b/src/data/drug-data.tsx
index 514742ba810b66195902f0953a9b6531b96be4ae..d1a3d6e6ab3da0307dfb08f373430ae786c7f7f3 100644
--- a/src/data/drug-data.tsx
+++ b/src/data/drug-data.tsx
@@ -46,11 +46,11 @@ export const drugdata: (Array<DrugDatensatz>)  = [
     {
         name: "Modulators",
         picture: "...",
-        introduction: "",
+        introduction: "CFTR modulators represent a significant advancement in CF treatment since they are small molecules improving the function of the defective CFTR protein in a mutation-specific way, which helps restore chloride ion transport across cell membranes. Notable pharmaceutical agents include Trikafta®, Symdeko®, Orkambi® and Kalydeco® [1]. These medications have been demonstrated to significantly improve lung function and reduce pulmonary exacerbations. However, they are expensive and may cause side effects such as liver enzyme elevations and cataracts in pediatric patients [2]. Furthermore, they are not suitable for all CF patients since only mutations which produce a CFTR channel can be supported by CFTR modulators, not those mutations which lead to a missing CFTR channel (knock out) [1], e.g. stop-mutations including p.Arg553Ter or p.Gly542Ter [3]. ",
         examples: [
             {
                 title: "",
-                text: "string"
+                text: "test"
             },
             {
                 title: "",