diff --git a/src/App/Timelines.css b/src/App/Timelines.css
index 2b2ad3acde52553767b7303143d961c1a64aeb9e..c14380430da3e9d637f5566f5e898fbc7c863bb3 100644
--- a/src/App/Timelines.css
+++ b/src/App/Timelines.css
@@ -367,4 +367,11 @@ display: flex;
 
 .timeline-item figcaption, .timeline-item figure, .timeline-item figcaption h3 {
   background-color: white !important;
+}
+
+
+
+.timeline-item .img-cube{
+  height: 120px;
+  max-height: 13vh;
 }
\ No newline at end of file
diff --git a/src/contents/description.tsx b/src/contents/description.tsx
index 9ed55a215a8c68c181b976a4d043ce5a46558784..9973655ded76d524f5a8fc78081ac3947925cf6b 100644
--- a/src/contents/description.tsx
+++ b/src/contents/description.tsx
@@ -34,7 +34,7 @@ export function Description() {
                         <div className="col" >
                             <img className="img" src="https://static.igem.wiki/teams/5247/placeholders/placehilderperson.jpeg"/> 
                         </div>    
-                    </div> 
+                    </div>
                     <p>By focusing on the F508del mutation, we also hope to contribute valuable insights to the global cystic fibrosis community. Although this mutation is most common in European populations, it is also found in other regions around the world [4, 5]. Our research could thus help inform treatment strategies and health policies on an international scale. </p>
                     <p>With several team members focusing their studies on biomedical fields, we began by examining the current landscape of CF treatments. It quickly became clear that, despite recent progress, there is still no cure. Most therapies, such as CFTR modulators, focus on managing symptoms and improving lung function rather than addressing the underlying cause of the disease [6]. This realization led us to explore gene-editing technologies, thus leading us to Prime Editing—a next generation gene editing method—captured our attention. </p>
                     <p>While Prime Editing holds great promise, we found that its application for cystic fibrosis, particularly the F508del mutation, had not been fully explored. Recognizing this gap in the research inspired us to take on the challenge of optimizing Prime Editing for this specific mutation. Our mission became clear: we want to contribute to the development of a potential therapeutic approach for cystic fibrosis, specifically targeting the F508del mutation with prime editing, and bring us closer to a long-term solution for patients. </p>
@@ -142,6 +142,12 @@ export function Description() {
                         </div> 
                     </Collapsible>  
                     <p>Overall, the statistical distribution of CFTR mutations reveals significant variations in mutation types and their frequencies worldwide, with deletions (72.64%) being the most common mutation type. This underscores the need for continued research and monitoring of these genetic variations to improve patient care and treatment strategies. CF not only affects the directly affected organs, but also many other areas of the body that are indirectly affected by the extent of the disease, e.g. through the condition of diseased organs. </p>
+                    <div className="row">
+                        <div className="col">
+                            <PieChart/>
+                        </div>
+                        <div className="col"></div>
+                    </div>
                 </Subesction>
                 <Subesction title="Symptoms" id="Cystic Fibrosis4">
                     <p>Since the CFTR gene is expressed in nearly all tissues of the human body, cystic fibrosis affects as a metabolic disease a wide range of vital organs.</p>